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Could fire be the key to slowing the spread of CWD?

Discussion in 'Wildlife Diseases' started by RMH, Jun 26, 2017.

  1. stickbow shooter

    stickbow shooter

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    Like getting rid of food plots ?
     

  2. Luv2hunteup

    Luv2hunteup

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    It may be spread by rumors too.
     
    Cork Dust likes this.
  3. FREEPOP

    FREEPOP

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    Destroying the habit will cause animals to disperse and will help spread the disease.

    At best it will only delay the inevitable, it could even accelerate it.
     
    Last edited: Jun 27, 2017
    terry likes this.
  4. Joe Archer

    Joe Archer Staff Member Mods

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    Didn't an enclosure once try incineration and find that CWD was still present when regrowth was allowed?
    <----<<<
     
  5. FREEPOP

    FREEPOP

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  6. terry

    terry

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    joe, i remember something about and old study in England, never had documented (not burning but burial of scrapie), just internal communication with someone on the inside i called deep throat. but i think this idea of torching say the state of Wisconsin, because of CWD, would be futile, and like the Freepop said, could even ''accelerate it'', by scattering all the animals and small critters that have been exposed to cwd tse prion.

    Freepop stated;

    Destroying the habit will cause animals to disperse and will help spread the disease.

    At best it will only delay the inevitable, it could even accelerate it.

    ===

    but you and others might be interested in the old BSE science back in the BSE Inquiry days, see where they were discussing this with trying to get rid of BSE cattle carcasses....you might find interest in this...

    http://scrapie-usa.blogspot.com/2011/02/environmental-sources-of-scrapie-prions.html

    Thursday, February 17, 2011

    Environmental Sources of Scrapie Prions

    SNIP...

    Some unofficial information from a source on the inside looking out -

    Confidential!!!!

    As early as 1992-3 there had been long studies conducted on small pastures containing scrapie infected sheep at the sheep research station associated with the Neuropathogenesis Unit in Edinburgh, Scotland. Whether these are documented...I don't know. But personal recounts both heard and recorded in a daily journal indicate that leaving the pastures free and replacing the topsoil completely at least 2 feet of thickness each year for SEVEN years....and then when very clean (proven scrapie free) sheep were placed on these small pastures.... the new sheep also broke out with scrapie and passed it to offspring. I am not sure that TSE contaminated ground could ever be free of the agent!! A very frightening revelation!!!

    ----------

    You can take that with however many grains of salt you wish, and we can debate these issues all day long, but the bottom line, this is not rocket-science, all one has to do is some experiments and case studies. But for the life of me, I don't know what they are waiting on?

    Kind regards,

    Terry S. Singeltary Sr. Bacliff, Texas USA

    More here:

    http://collections.europarchive.org...ttp://www.bseinquiry.gov.uk/files/ws/s018.pdf

    http://chronic-wasting-disease.blogspot.in/2011_12_01_archive.html

    Using in vitro prion replication for high sensitive detection of prions and prionlike proteins and for understanding mechanisms of transmission.

    Claudio Soto Mitchell Center for Alzheimer's diseases and related Brain disorders, Department of Neurology, University of Texas Medical School at Houston.

    ***Recently, we have been using PMCA to study the role of environmental prion contamination on the horizontal spreading of TSEs. These experiments have focused on the study of the interaction of prions with plants and environmentally relevant surfaces. Our results show that plants (both leaves and roots) bind tightly to prions present in brain extracts and excreta (urine and feces) and retain even small quantities of PrPSc for long periods of time. Strikingly, ingestion of prioncontaminated leaves and roots produced disease with a 100% attack rate and an incubation period not substantially longer than feeding animals directly with scrapie brain homogenate. Furthermore, plants can uptake prions from contaminated soil and transport them to different parts of the plant tissue (stem and leaves). Similarly, prions bind tightly to a variety of environmentally relevant surfaces, including stones, wood, metals, plastic, glass, cement, etc. Prion contaminated surfaces efficiently transmit prion disease when these materials were directly injected into the brain of animals and strikingly when the contaminated surfaces were just placed in the animal cage. These findings demonstrate that environmental materials can efficiently bind infectious prions and act as carriers of infectivity, suggesting that they may play an important role in the horizontal transmission of the disease.

    https://prion2015.files.wordpress.com/2015/05/programguide1.pdf

    http://www.tandfonline.com/doi/pdf/10.4161/pri.28467

    In conclusion, the results in the current study indicate that removal of furniture that had been in contact with scrapie-infected animals should be recommended, particularly since cleaning and decontamination may not effectively remove scrapie infectivity (31), even though infectivity declines considerably if the pasture and the field furniture have not been in contact with scrapie-infected sheep for several months. As sPMCA failed to detect PrPSc in furniture that was subjected to weathering, even though exposure led to infection in sheep, this method may not always be reliable in predicting the risk of scrapie infection through environmental contamination. These results suggest that the VRQ/VRQ sheep model may be more sensitive than sPMCA for the detection of environmentally associated scrapie, and suggest that extremely low levels of scrapie contamination are able to cause infection in susceptible sheep genotypes.

    Keywords: classical scrapie, prion, transmissible spongiform encephalopathy, sheep, field furniture, reservoir, serial protein misfolding cyclic amplification

    http://journal.frontiersin.org/article/10.3389/fvets.2015.00032/full

    Wednesday, December 16, 2015

    *** Objects in contact with classical scrapie sheep act as a reservoir for scrapie transmission ***
    http://scrapie-usa.blogspot.com/2015/12/objects-in-contact-with-classical.html

    *** Infectious agent of sheep scrapie may persist in the environment for at least 16 years ***
    Gudmundur Georgsson1, Sigurdur Sigurdarson2 and Paul Brown3

    http://jgv.sgmjournals.org/content/87/12/3737.full

    New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication

    http://www.pnas.org/content/97/7/3418.full

    Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production

    http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2493038/

    Detection of protease-resistant cervid prion protein in water from a CWD-endemic area

    http://www.landesbioscience.com/journals/prion/NicholsPRION3-3.pdf

    kind regards, terry
     
  7. terry

    terry

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    more from deep throat;

    DEEP THROAT TO TSS 2000-2001 (take these old snips of emails with how ever many grains of salt you wish. ...tss)

    The most frightening thing I have read all day is the report of Gambetti's finding of a new strain of sporadic cjd in young people...Dear God, what in the name of all that is holy is that!!! If the US has different strains of scrapie.....why????than the UK...then would the same mechanisms that make different strains of scrapie here make different strains of BSE...if the patterns are different in sheep and mice for scrapie.....could not the BSE be different in the cattle, in the mink, in the humans.......I really think the slides or tissues and everything from these young people with the new strain of sporadic cjd should be put up to be analyzed by many, many experts in cjd........bse.....scrapie Scrape the damn slide and put it into mice.....wait.....chop up the mouse brain and and spinal cord........put into some more mice.....dammit amplify the thing and start the damned research.....This is NOT rocket science...we need to use what we know and get off our butts and move....the whining about how long everything takes.....well it takes a whole lot longer if you whine for a year and then start the research!!! Not sure where I read this but it was a recent press release or something like that: I thought I would fall out of my chair when I read about how there was no worry about infectivity from a histopath slide or tissues because they are preserved in formic acid, or formalin or formaldehyde.....for God's sake........ Ask any pathologist in the UK what the brain tissues in the formalin looks like after a year.......it is a big fat sponge...the agent continues to eat the brain ......you can't make slides anymore because the agent has never stopped........and the old slides that are stained with Hemolysin and Eosin......they get holier and holier and degenerate and continue...what you looked at 6 months ago is not there........Gambetti better be photographing every damned thing he is looking at.....

    Okay, you need to know. You don't need to pass it on as nothing will come of it and there is not a damned thing anyone can do about it. Don't even hint at it as it will be denied and laughed at.......... USDA is gonna do as little as possible until there is actually a human case in the USA of the nvcjd........if you want to move this thing along and shake the earth....then we gotta get the victims families to make sure whoever is doing the autopsy is credible, trustworthy, and a saint with the courage of Joan of Arc........I am not kidding!!!! so, unless we get a human death from EXACTLY the same form with EXACTLY the same histopath lesions as seen in the UK nvcjd........forget any action........it is ALL gonna be sporadic!!!

    And, if there is a case.......there is gonna be every effort to link it to international travel, international food, etc. etc. etc. etc. etc. They will go so far as to find out if a sex partner had ever traveled to the UK/europe, etc. etc. .... It is gonna be a long, lonely, dangerous twisted journey to the truth. They have all the cards, all the money, and are willing to threaten and carry out those threats....and this may be their biggest downfall...

    Thanks as always for your help. (Recently had a very startling revelation from a rather senior person in government here..........knocked me out of my chair........you must keep pushing. If I was a power person....I would be demanding that there be a least a million bovine tested as soon as possible and agressively seeking this disease. The big players are coming out of the woodwork as there is money to be made!!! In short: "FIRE AT WILL"!!! for the very dumb....who's "will"! "Will be the burden to bare if there is any coverup!"

    again it was said years ago and it should be taken seriously....BSE will NEVER be found in the US! As for the BSE conference call...I think you did a great service to freedom of information and making some people feign integrity...I find it scary to see that most of the "experts" are employed by the federal government or are supported on the "teat" of federal funds. A scary picture! I hope there is a confidential panel organized by the new government to really investigate this thing.

    You need to watch your back........but keep picking at them.......like a buzzard to the bone...you just may get to the truth!!! (You probably have more support than you know. Too many people are afraid to show you or let anyone else know. I have heard a few things myself... you ask the questions that everyone else is too afraid to ask.)

    SATURDAY, JULY 15, 2017

    National Prion Center could lose all funding just as concern about CWD jumping to humans rises

    http://creutzfeldt-jakob-disease.blogspot.com/2017/07/national-prion-center-could-lose-all.html

    http://creutzfeldt-jakob-disease.blogspot.com/2016/08/creutzfeldt-jakob-disease-usa-2015.html
     
  8. terry

    terry

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